Beyond being aware that they are important, do you, like me, have only the sketchiest idea of what your mitochondria are or do? If so, you need to log in to Biolab’s Mitochondria day on September 17th when a range of mitochondria experts will not only explain what they are, but what their relationship could be to obesity, CFS/ME, MS, Parkinsons – and even dementia. And, most importantly, how treatment can improve patient outcomes.
To give you a brief heads up – thanks to the Mitochondrial Biology Unit:
Every living thing is made of cells, tiny compartments contained by a membrane. Each cell is made up of organelles, one of which are mitochondria, and they all perform different functions that are essential for the cell to live. The mitochondria produce energy; in fact they produce about 90% of the chemical energy that cells need to survive.
The mitochondria also produce chemicals that the body uses to break down waste products and recycle some of them to save energy. And they have a special role in making cells die (apoptosis), crucial for the processes of growth and development. When cells don’t die as they should, they can start to grow uncontrollably creating a cancerous tumour.
What will the day cover?
An introduction to mitochondria, how they function, what can go wrong and how you can test for that.
Research into the connection betweeen mitochondrial malfunction and:
- Chronic disease states
- Chronic Fatigue Syndrome, ME and fibromylagia
- Multiple Sclerosis and dementia
- Parkinson’s Disease
- Dr Sarah Myhill
- Professor Eija Pirinen
- Dr Jenny Goodman
- Lucille Leader
- Mark Adams
- Gilian Crowther
- Rachel Nicholl
Or for full details on the speakers spool down the Eventbrite booking page.
You can attend in person or join on zoom and, if you book before the end of August, you will get a 30% early bird discount.